Digestive Tract Development in Newborn Calves

Most newborn calves are separated from their mother soon after birth on modern dairy farms. In the first few weeks of life they then consume less milk solids daily than if they were nursed by their mothers. We then expect rapid growth and development of the GI tract to accommodate digestion of solids feeds and early weaning. Natural weaning takes up to ten months. We expect our calf to achieve this in two months.

Enormous research has gone into calf rearing systems to minimise cost and labour rather than focusing on development of the GI tract to digest solid feeds. Right or wrong, our new born calf is certainly “put to the test” during its first few weeks of life. Despite history telling us through ever increasing productivity of our cows we’re doing a better job, however, USDA data highlights mortality rate is still around 10% and morbidity (depressed) near 50%.

There are two challenges a newborn must overcome: 1) they must overcome attacks on their immune systems, 2) transition from a monogastric to a ruminant. Weaning at two months is determined by surviving a pathogen attack in the first few weeks. Digestive disease is number one threat. Gut ailments or infections either end in mortality or expensive antibiotic treatment costs and retard development. Calves are born with little immunity and we have twelve hours to administer colostrum for it to be effective to passive immune transfer.

Even the switch to whole milk or milk replacer immediately after administration of colostrum, robs our calf of hormones, prebiotics and immune stimulants that may aid in development of the GI tract, and are found in fresh cow’s milk for up to a week after calving. There is a strong argument for collecting fresh cow milk for a week post-calving for feeding calves. It is also higher in fats and protein than milk past the first week. If you are using milk replacers, ensure they contain milk fats and proteins avoiding products containing fats and proteins of vegetable origin; they are poorly digested.

A calf should be drinking milk with 12% to 13% solids and at a minimum rate of 10% of their body weight. Calves fed up to 20% of their body weight have been shown to have higher lifetime milk production, but requires multiple daily feedings to avoid gut disease/infection (refer last month’s ‘Rumen Drinking’ article). While being fed as a monogastric on milk, the rumen is rapidly developing. At birth the calf rumen is only 25% of the forestomach. It must reach 70% to enable sound growth on solid feeds. Coarse calf grain mix is most effective for developing the rumen, enabling transformation of our calf’s diet from calories and energy from milk to Volatile Fatty Acids (VFA) from ruminal fermentation. Allowing 4 or 5 kgs of grain consumption post-weaning will accommodate the calf’s need for VFA’s for sustained growth, health and further development of the rumen to allow for digestion of forage which is very limited before six months of age. They will not get acidosis.

Newborn calves that survive pathogenic challenges will naturally develop a functioning rumen and should double their birth weight in sixty days. There is as much art in this as science, as any successful calf rearing person knows.

CALF SCOURS & Misconceptions

Misconception #1 Nutritional scours is a common problem in calves

A generation or two ago, nutritional scours was an issue, but today’s high quality/low cell count milk and calf powdered milks no longer carry scour causing organisms common in the past. Nutritional scours were diagnosed on the basis calves passing high amounts of manure. A calf consuming 0.8 kgs (6 lts x 13% solids) of milk solids daily will pass significant amounts of manure. Nutritional scours can occur through stress. Milk changes, environmental, transport, vaccination, weather, dehorning etc. However, these stress induced scours usually pass in a couple of days.

Misconception #2 Liquid manure is scours

As above, high milk intakes common today will produce liquid manure. Calves that are healthy, active and showing no signs of dehydration are unlikely to have scours.

Misconception #3 Electrolytes don’t work

There are electrolytes on the market that are very light-on in minerals and glucose. Perhaps veterinary advice is warranted here. Quality electrolytes are very effective in rehydration and mineral support when used correctly. They must be fed in between milk feedings as the water content is as important as the minerals.

Misconception #4 Type of scour can be identified by colour

Rotavirus, coronavirus and even increased milk can produce a white scour. Several bacterial strains can produce the same colour scour. Fecal culture is the only way to identify the type of scour.

Misconception #5 Scour type can be identified by calf age

It is true some diseases can occur based on days from birth (eg salmonella, e. coli etc). However, if the calf has a true scour, and not just high volume manure from a high plain of nutrition, then that can happen at any age.

Misconception #6 Reduce feed intake for sick calves

Many years ago the rule was ‘No milk while using electrolyte’. Most calves still died. Why? Starvation! Their energy requirement increases to support an energy-hungry immune system under challenge. Possibly, what had been said, was not to feed milk and electrolytes together.

Environmental hygiene goes without saying. There are a number of products on the market for disinfecting calf sheds. However, the most effective product we have witnessed is Vibrex. (Available from Roy Watson 0428 526 581 )

Ensuring calf nutrition is optimal, that is, timely administration of quality colostrum, that they are receiving adequate milk solids daily (fresh milk can vary – it pays to check – see Feb 20th article on refractometers), add to this CALFMAX containing essential minerals, vitamins, Bovatec, MOS, glucans and galactosamine, and disease and scours can largely be avoided.

Limestone and Salt

Perhaps the two most abundant compounds on the face of the earth, yet our cows are deficient in both, with significant implications to health, fertility and milk production. Why, perhaps due to their commonness, and we tend to take little notice of things that are very common.

Salt is an essential compound in a cow’s diet for a number of metabolic functions, but also for buffering capacity of saliva in the rumination process. Apart from these important functions, salt will make cows drink water, and there is a direct correlation between water intake and dry matter intake; which naturally drives milk production. Salt should be in the dairy grain mix, but also offered free choice as the cows’ needs can vary daily.

The subject I really want to address is calcium in our cow’s diet. Limestone dressing on pastures is another subject that requires urgent attention. No, or low, calcium in soils will seriously retard plant growth and mineral content of plants. Calcium is the transport system of other minerals in both our soil and our cows’ rumens.

Gary Oetzel, DVM, professor of dairy science at the University of Wisconsin, during a seminar stated: “You ought to respect hypocalcemia, you ought to be afraid of it, and you ought to do everything you can to minimise it”. Hypocalcemia is low blood calcium. Milk Fever in clinical form, but we’ve made massive inroads on milk fever with anionic lead feeds and good transition management reducing incident rates to 2% – 3% of the herd. Despite this, sub-clinical milk fever is reported to run around 60% of all second-plus lactation cows. Heifers are not immune either.

The clinical milk fever cow can be recognised, treated, and back in production relatively easily. The consequences of sub-clinical hypocalcaemia have far more serious effects than we realise, and far more reaching into lactation production, health and fertility. Blood tests are the only real indicator of the degree of hypocalcaemia. Initially, at calving, there are big benefits in oral calcium supplements (drenches or boluses) to help cows cope with “lactational osteoporosis”. This is virtually unavoidable to meet the 300% increase in calcium that fresh cows require. Oetzel estimates sub-clinical hypocalcaemia at four times the cost of clinical milk fever. When research has identified up to 60% of mature cows suffer sub-clinical hypocalcaemia, at four times the cost of clinical milk fever, then this is a very significant cost to farm profit.

Sub-clinical hypocalcaemia is well documented as the prerequisite to a numerous diseases common around calving: obviously clinical milk fever, but add to that, retained membrane, metritis, mastitis, ketosis, and a major industry problem, fertility. Productive life as milking cows can be unnecessarily cut short as a result of sub-optimal calcium nutrition. Our clients that have been proactive in calcium nutrition have seen major decreases in fresh cow mastitis, metritis, and very significant improvements in fertility.

My concern is, in our low calcium ryegrass pastures, this hypocalcemic disorder goes on well past early lactation. A cow producing 30 litres of milk is exporting 36 gm of calcium daily in milk. That’s the calcium content of 100+ gms of limestone. Then there are urinary loses, reabsorption of calcium into bones and metabolic functions requiring calcium. The immune system alone has a high calcium demand.

Dietary calcium deficiencies are common in ryegrass pastures which frequently have little or no clover content these days. Milk protein production is also limited by calcium deficient diets. Ryegrass contains typically around 0.3% calcium. Clover is around 1.4% and Lucerne 1.7%. A recent article in the Australian Dairyfarmer highlighted the need for a return to 30% to 40% clover content in grazing pastures. This article was more about pasture production and soil health and their need for clover, rather than cow nutrition.

Soils have a high need for calcium to function well (transport other nutrients). Obviously, legumes have a high need for soil calcium availability for growth, and their high calcium content. The author of the Dairyfarmer article stated “we’ve lost our way” in pasture production with the neglect of clover. Losing our way is correct. When I started share farming in the 1970’s, applying lime was an annual autumn event. Where I live in SW Victoria we sit on top of a limestone mountain, yet I see very little spreading of lime on pastures. Certainly SW Vic would benefit immensely from gypsum applications. Gypsum will supply sulphur as well as calcium, another element lacking in our soils as a legacy of high analysis fertilisers and urea. Gypsum will restore soil structure allowing for better water and air penetration.

I think the massive uptake of urea use, the demise of super applications, the spraying of broadleaf weeds have all contributed to clover’s departure from our pastures. These are all reversible including broadleaf sprays that are now available and harmless to clover. A return to lime applications must precede sowing of clover. It is not just a soil pH issue, but a locking up of other nutrients that only calcium can undo that needs to happen to ensure good clover establishment and perseverance.

Reestablishment of clover in our pastures will not just increase calcium in the diet of our cows with major benefits to production, health and fertility, but increase pasture harvested per hectare, improve soil nutrient availability to plants, improve soil structure and reduce soil compaction problems prevalent on most dairy farms.

I profess no expertise in agronomy and strongly advise consultation with sound agronomists. Calcium is not a stand-alone cure-all. It is intimately involved with numerous other elements in both soil and animal nutrition. However, calcium is called a macro element, meaning, it is required in large amounts and our capacity to meet this need through grain mixes alone is limited. Increasing calcium in our forages through good pasture clover content will go a long way in improving both pasture and cow health and production. As the Dairyfarmer article said; “we’ve lost our way”. This is not rocket science, but was standard, good-farming practice of 50 years ago.

Leafy summer fodder crops like sorghum have a high need for calcium and respond in yield very significantly when calcium is not a limitation. As one of my clients says; lime is a fertiliser.

Dry Matter Intake

In the words of Professor Mike Hutjens, University of Illinios, “Don’t give up on dry matter intake”. He says further, “High dry matter intake solves a lot of problems on a lot of farms”. I could not agree more!

Nothing reduces immediate profit, converting feed dollars to milk dollars, than underfeeding. Most common feeds in our dairy industry are capable to being converted from 1 kg DM of feed to 2 litres of milk after maintenance is met. The math is fairly simple. Even feed at 30c/kg DM (barley) will convert to at least 60c of milk income. Obviously energy density of that feed does influence conversion efficiency. Nevertheless, the lesson stands; if you want to increase profit dramatically, then offer more feed to your cows.

From 16 years of production consulting and 25 years of dairy farming, I can assure you the average Australian dairy cow is 4 kgs DM underfeed. That’s 8+ litres! By our conversion factor of 1 kg DM feed to 2 litres of milk this would produce $1/cow/day more profit. Our cows have incredible capacity to make profit from converting feed to milk.

So why didn’t our cow eat the extra feed?

1) Without doubt, she did not have access to the extra feed. This is by far the greatest limitation to profit in Australian dairy businesses. While the lactation average for the last twelve months in the USA was 10,450 litres and ours is 5200 litres, yet we hope to compete with the USA on world markets, I can assure you the simple difference is feed intake; bar none. The preparation of Feed Budgets for our clients is solely aimed at optimising milk production which has direct relationship to profit, and worse (or better), by a multiplier coefficient.

2) Her rumen is full. Isn’t this what we’re aiming for? Yes, if the feed is balanced for energy, protein and fibre. No if the fibre level is too high and it spends long periods of time in the rumen being digested. Indigestible fibre especially slows down the rate of passage of feed through the digestive tract reducing total feed intake; and milk production. The monthly ration analysis we do for all clients has the goal of a balanced and productive ration to optimise milk production under feed scenarios throughout the season. This is also tempered by what we grow to meet the Feed Budget.

3) The cow stops eating for no apparent reason. This usually occurs from metabolic signalling within the cow’s system. Too much rapidly fermented carbohydrate reducing rumen pH, either clinically or sub-clinical (SARA). Grain is not the only culprit here. It can be rapidly growing pasture with low fibre, frequently cows go from the dairy (grain) straight onto turnips which are highly digestible. Silage feed in between these two will solve a lot of digestive limitations to feed intake. Our pastures have risen in unsaturated fats in recent years through plant breeding. The rumen must convert these unsaturated fats to saturated fats before being transported to the mammary system. An excess of fat will reduce intake, reduce digestion of fibre and in high excess, will crash BF%. Mineral supplementation is essential for health, fertility, but also appetite and feed intake. Good transition nutrition will drive higher peak milk and appetite.

If you want to improve the viability of your dairy business, call Tina in our office (0400 991 814) and she will arrange a ‘no obligation’ visit by one of our team for to explain our nutrition/production services to you. As Mike Hutjens says, “Never give up on MILK!” and never forget your farm is a business.

Calf Water Intake is Dynamic

A 36 kg calf will lose around 3.8 lts of water from its body in urine, faeces, skin, mouth and respiratory secretions each day. 4 litres of milk intake only provides 3.5 lts of water/day. Without additional water intake over and above milk, the calf will dehydrate and most likely die from disease having easy domination over a water-stressed system.
Add to this, a sick calf with diarrhoea will drink less milk, excrete more water and usually dies of dehydration. If you are feeding electrolytes to scouring calves it is necessary they either drink milk also, or calf starter grain to maintain energy intake or starvation will finish them. Initially, until the calf is drinking from a separate water source, filling the milk feeder with warm water within 10 to 30 minutes of feeding milk will drive water intake. Water temperature does affect intake. Research has demonstrated a 50% increase in water intake when close to body temperature.
There is a direct correlation between water intake and calf grain intake. Research has suggested between 4:1 & 6:1 ratio of water to grain intake. Taking an average of 5:1, our calf must drink 5 litres of water to consume 1 kg of calf grain. A good calf grain mix should supply more energy and protein than milk. Obviously, the faster we have calves increasing water intake, the more grain they will eat and their plain of nutrition (energy & protein) will rise rapidly. Accordingly, growth rate will also accelerate rapidly. By day 56 this ratio is closer to 2:1 through rumen development.
Calf grain intake plays another major role apart from simply increasing energy and protein intake. A course grain mix (as opposed to pelleted) will help increase water intake, but apart from that, starch from grain will produce Volatile Fatty Acids from grain digestion which drives the growth of rumen papillae, the absorption site of nutrient to the blood stream. The faster we develop rumen papillae the sooner the calf can be weaned as it will be capable of digestion solid foods then. We use calf grain intake as the indicator for weaning. A calf consuming 2 kgs of calf grain daily has developed the rumen sufficiently to digest solid feeds and can be weaned. There is a significant cost saving in both feed cost and labour to benefit from by early weaning.
Grain intake must continue to rise post-weaning as the rumen, although developed sufficiently to extract nutrient from grain mixes, it still cannot digest forage adequately to supply the necessary energy and protein for both maintenance and growth. Calves are unable to digest forage well before six months of age. Water intake remains the driver of post-weaning growth by its ratio of 2:1, water to grain, increasing the calf’s rising plane of nutrition correlating growth and development. Water quality also plays a major role in calf growth due to its relation to grain intake.